Description
5-MeO-MiPT is a tryptamine. The psychedelic 5-meo-dmt is a homologue. 5-methoxy-N-methyl-N-isopropyltryptamine is the full name of this chemical.
Also, it causes the ehrlich reagent to turn purple then fade to faint blue. It causes the marquis reagent to go yellow through to black.
Effects of 5-meo-mipt
It has the nickname “moxy” and it is an analogue of the more popular drug 5-MeO-DiPT . Also, 5-MeO-DiPT is nicknamed “foxy methoxy”. However, some users report the tactile effects of 5-MeO-DiPT without some of the unwanted side effects.It becomes much more psychedelic at higher doses. It sometimes compared to 5-MeO-DMT.
However, at doses of 4-10 milligrams users find 5-MeO-MiPT to be a very euphoric and tactile chemical. Its energetic effects can be very strong at high doses, increasing normal heart rate considerably. Sounds can be amplified in perception to a point where synesthetic effects (“touching or/and tasting sounds”) occur.
Dosage
Orally, 5-MeO-MiPT is active at 4-6 mg. The drug can be smoked, but requires a much higher dosage. This, 10–20 mg is usually smoked. It typically produces a very strong odor.
Some users report activity as low as 1 mg while others report no activity up to 20 mg. However, 5-meo-mipt seems to be highly sensitive to the individual and any potential researchers should keep this in mind. Titrating the dose would be especially important with this compound.
Little to no visual activity reported by users at 10 mg or higher doses. This chemical proves very useful for opening up and expressing oneself much like MDMA. Lastly, it may be a useful chemical in psychedelic therapy.
Pharmacology
Mechanism producing the hallucinogenic and entheogenic effects of 5-MeO-MiPT result primarily from 5-HT2A receptor agonism. Also, additional mechanisms of action such as inhibition of MAO may be involved also. 5-MeO-MiPT binds most strongly to 5-HT1A receptors; it also shows fairly strong binding affinity to the SERT and NET, thereby acting as a moderately potent serotonin-norepinephrine reuptake inhibitor. These mechanisms may help explain why there are many anecdotal reports of anti-depressant and anxiolytic effects from modest doses of this compound. SNRIs such as venlafaxine used to treat depression.
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